The hereditary dentine disorders, dentinogenesis imperfecta (DGI) and dentine dysplasia (DD), comprise a group of Review; Open Access. clinical section. Dentinogenesis imperfecta: an early treatment strategy Dentinogenesis imperfecta (DI) type 2 is a disease inherited in .. prehensive review. PDF | Dentinogenesis imperfecta (DI) is one of the most common hereditary disorders of dentin formation. It follows an autosomal dominant.

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Further studies, for certain, are extremely required to finally elucidate the outlines of this dramatic dental condition.

Management of dentinogenesis imperfecta: a review of two case reports.

Cells Tissues Organs,pp. Oral rehabilitation in dentinogenesis imperfecta with overdentures. A proposed classification for heritable human dentine defect with a description of a new entity. J Biol Chem,pp. Rajendran R, Sivapathasundram B, editors. A proposed classification for heritable human dentine defects with a description of a new entity. A text book of oral pathology, WB Saunders Co, Arch Oral Biol, 18pp.

Dentinogenesis imperfecta in the Brandywine isolate: A review and case report of a family over four generations. Continuing navigation will be considered as acceptance of this use.

J Dent Res, 86pp. Dentinogenesis is a highly controlled process that results in the encompassed mineralization of the predentin into a mineralized matrix. You can change the settings or obtain more information by clicking here.


Dentinogenesis imperfecta type I: A case report with literature review on nomenclature system.

RGO, 48pp. Dentinogenesis imperfecta type III with enamel and cementum defects. Dentinogenesis imperfecta DGI is one of the most common hereditary disorders of dentin formation.

J Biol Chem ; Autosomal dominant, dentinogenesis imperfecta, dystrophic dentin, mesodermal defect, pulpal space obliteration. Developmental disturbances of oral and paraoral structures.

CiteScore measures average citations received per document published. Intrafibrillar mineral may be absent in dentinogenesis imperfecta type II. Previous article Next article.

To understand the molecular basis of this disorder, as well as, its clinical and radiographic evidences, it is essential to make an appropriate diagnosis and ulterior treatment. Dentinogenesis Imperfecta DI represents one of those abnormalities inherited in a dominant autosomic pattern.

Professor Auxiliar na Universidade Fernando Pessoa. Discontinued publication For more information click here.

Dentinogenesis imperfecta type I: A case report with literature review on nomenclature system.

Acta Odontol Scand ; Cloning and sequence determination of rat dent sialoprotein, a novel dentin protein. J Dent Child, 48pp. J Oral Pathol ; Unusual dentinal changes in dentinogenesis imperfecta associated with osteogenesis imperfecta.

Int J Pediatr Dent ;9: Any interference during the mineralization phase may affect the dentin final structure, which depending on the severity of the disturbance involved, may be detected in both dentitions, deciduous and permanent.


To improve our services and products, we use “cookies” own or third parties authorized to show advertising related to client preferences through the analyses of navigation customer behavior. How to cite this article: The non collagenous dentin matrix proteins are involved in dentinogenesis imperfecta type II. Dentin phosphoprotein DNA sequence determination. The main goal of this literature review is to highlight the genetic aspects that underline its establishment and to report the most prevalent clinical and radiographic clues present in reviee patient with DI.

Discrimination of morphological findings in osteogenesis imperfecta patients using combination of polarized light microscopy, microradiography and scanning electron microscopy. J Oral Pathol Med, 34pp. J Am Dent Assoc ; Revview Dent Res, 85pp.

Scanning electron microscopy of teeth in osteogenesis imperfecta type I. Under a Creative Commons license. Scopus See more Follow us: Tatiana Cardoso a ,??

Acta Odontol Scand, 61pp.

Professor Associado na Universidade Fernando Pessoa.